Keywords

• Inosine
• Testosterone Base
• Testosterone Acetate

Quick Details

• ProName: Inosine Coenzyme drug raw material
• CasNo: 58-63-9
• Molecular Formula: C26H40O3
• Appearance: White powder
• Application: Inosine CAS：58-63-9 Product Name: ...
• DeliveryTime: 7days
• PackAge: Foil bag
• ProductionCapacity: 50000 Kilogram/Week
• Purity: 99
• Storage: Placed in a cool place
• LimitNum: 1 Gram
• Moisture Content: 0.001
• Impurity: 0.001

Superiority

Details

Inosine 

CAS：58-63-9

Product Name:    Inosine

Synonyms:    Atorel;beta-D-Ribofuranoside, hypoxanthine-9;beta-Inosine;HXR;Hypoxanthine D-riboside;Hypoxanthine, 9-beta-D-ribofuranosyl-;hypoxanthined-riboside;hypoxanthinenucleoside

CAS:    58-63-9

appearance:White crystalline powder

Assay:99%

MF:    C10H12N4O5

MW:    268.23

EINECS:    200-390-4

Product Categories:    Nucleotides and Nucleosides;Antivirals for Research and Experimental Use;Biochemistry;Nucleosides and their analogs;Nucleosides, Nucleotides & Related Reagents;Nucleic acids;Bases & Related Reagents;Nucleotides;Inhibitors

Inosine is a nucleoside that is formed when hypoxanthine is attached to a ribose ring (also known as aribofuranose) via a β-N9-glycosidic bond.

Inosine is commonly found in tRNAs and is essential for proper translation of the genetic code in wobble base pairs.

Knowledge of inosine metabolism has led to advances in immunotherapy in recent decades. Inosine monophosphate is oxidised by the enzyme inosine monophosphate dehydrogenase, yielding xanthosine monophosphate, a key precursor in purine metabolism. Mycophenolate mofetil is an anti-metabolite, anti-proliferative drug that acts as an inhibitor of inosine monophosphate dehydrogenase. It is used in the treatment of a variety of autoimmune diseases including granulomatosis with polyangiitis because the uptake of purine by actively dividing B cells can exceed 8 times that of normal body cells, and, therefore, this set of white cells (which cannot operate purine salvage pathways) is selectively targeted by the purine deficiency resulting from inherited metabolic diseases (IMD) inhibition.

 

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